# STOCKHOLM 1.0
#=GF ID WT1-AS_5
#=GF AC RF02207
#=GF DE WT1 antisense RNA conserved region 5
#=GF AU Eberhardt R; 0000-0001-6152-1369
#=GF GA 80.0
#=GF NC 56.8
#=GF TC 87.4
#=GF SE Predicted; CMfinder
#=GF SS Predicted; CMfinder
#=GF TP Gene; lncRNA;
#=GF BM cmbuild -F CM SEED
#=GF CB cmcalibrate --mpi CM
#=GF SM cmsearch --cpu 4 --verbose --nohmmonly -T 21.44 -Z 2958934 CM SEQDB
#=GF DR SO; 0001463; lincRNA;
#=GF RN [1]
#=GF RM 2173145
#=GF RT Tissue, developmental, and tumor-specific expression of divergent
#=GF RT transcripts in Wilms tumor.
#=GF RA Huang A, Campbell CE, Bonetta L, McAndrews-Hill MS, Chilton-MacNeill S,
#=GF RA Coppes MJ, Law DJ, Feinberg AP, Yeger H, Williams BR
#=GF RL Science. 1990;250:991-994.
#=GF RN [2]
#=GF RM 8406502
#=GF RT Sequence of the WT1 upstream region including the Wit-1 gene.
#=GF RA Gessler M, Bruns GA
#=GF RL Genomics. 1993;17:499-501.
#=GF RN [3]
#=GF RM 17210670
#=GF RT A CTCF-binding silencer regulates the imprinted genes AWT1 and WT1-AS and
#=GF RT exhibits sequential epigenetic defects during Wilms' tumourigenesis.
#=GF RA Hancock AL, Brown KW, Moorwood K, Moon H, Holmgren C, Mardikar SH,
#=GF RA Dallosso AR, Klenova E, Loukinov D, Ohlsson R, Lobanenkov VV, Malik K
#=GF RL Hum Mol Genet. 2007;16:343-354.
#=GF WK WT1-AS
#=GF SQ 4

AL049692.13/51819-51741 GGCACUUUUAGCGGGGGGAGAGGAGGUUAUCUGAAGGGA----GGCUGGCUUGAAUGUGAGUCGUUGUGUUUGUGAAACCCAG
L25110.1/3560-3638      GGCACUUUUAGCGGGGGGAGAGGAGGUUAUCUGAAGGGA----GGCUGGCUUGAAUGUGAGUCUUUGUGUUUGUGAAACCCAG
X77549.1/328-250        GGCACUUUUAGCGGGGGGAGAGGAGGUUAUCUGAAGGGA----GGCUGGCUUGAAUGUGAGUCGUUGUGUUUGUGAAACCCAG
AANU01216576.1/178-102  GGCCCUUUUAGCGGGGGGAGAGGAGGUUAUCUGAAGGGAGGGAGGCAGGCUUGAAUGUGAGUC------UUUGUGAAACCCAG
#=GC SS_cons            :::::::::::::::::::::::::::::::::::::::....::::::::::::::::::::::::::::::::::::::::
#=GC RF                 GGCaCUUUUAGCGGGGGGAGAGGAGGUUAUCUGAAGGGA....GGCuGGCUUGAAUGUGAGUCgUUGUGUUUGUGAAACCCAG
//
